A síndrome de Legg-Calvé-Perthes ou doença de Perthes é uma doença degenerativa da articulação da anca (quadril em português brasileiro) em que ocorre. El absceso del psoas es una enfermedad infrecuente en niños. Puede tener una en pediatría es primario; sin embargo, en ocasiones, puede ser de origen secundario y médula espinal, enfermedad de. Legg-Calvé-Perthes, entre otros Download Citation on ResearchGate | Enfermedad de Legg Calvé Perthes (LCP) en varios aspectos de la enfermedad de Legg Calvé Perthes y la evolución de caderas de August · Anales espanoles de pediatria.
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Background It has been suggested that the cause of Perthes diseasemay be intravascular thrombosis induced by a potentialcongenital hemostatic disorder leading to conditions ofthrombophilia or hyperfibrinolysis.
Necesidades del alumnado con diabetes tipo 1 en The remaining patients were considered withinthe normal range when age was taken into account.
Antithrombin heparin cofactor assay with new chromogenic substrates. Thromb Haemost, 62pp. Clin Orthop,pp. Special laboratory evaluation prdiatria coagulation. N Engl J Med,pp. Disorders of hemostasis in childhood: Identification of the same factor V gene mutation in 47 of 50 thrombosis-prone families with inherited resistance to activated protein C.
Acta Med Scand,pp. Perthes’ disease and the relevance of thrombophilia.
Thromb Res, 11pp. Si continua navegando, consideramos que acepta su uso. Anal Biochem,pp.
Enfermedad de Legg-Calve-Perthes
Relation of altered hemostasis to etiology. Continuing navigation will be considered as acceptance of this use. J Biol Chem,pp.
Changes in coagulation-fibrinolysis system in Legg-Perthes disease: Variability of thrombosis among homozigous siblings with resistance to activated protein C due to pefiatria Arg-Gln mutaion in gen for factor V. Epidemiological and laboratory enfermexad from this group ofpediatric patients and from the control group do not supportthe hypothesis that a thrombogenesis defect couldbe the underlying cause of avascular necrosis of the hipjoint.
Am J Hematol, 45pp. Thromb Haemost, 69pp. Familial hypofibrinolysis and venous thrombosis.
It has been suggested that the cause of Perthes diseasemay be intravascular thrombosis induced by a potentialcongenital hemostatic disorder leading to conditions ofthrombophilia or hyperfibrinolysis. Pediatfia of antithrombotic factor deficiencies and hy-pofibrinolysis with Legg-Perthes disease. You can change the settings or obtain more information by clicking here.
J Rnfermedad Clin Med,pp. Results Alterations in hemostasis and thrombosis were detectedin one patient who had moderate-to-light protein Sdeficiency. Patients and method Complete study of hemostasis with coagulation and antithrombinfactors as well as study of thrombophilia andfibrinolysis in these patients could determine the prevalenceof hemostasis and fibrinolysis in this group of patients.
Blood, 85pp. Maturation of the hemostatic system during childhood. Am J Clin Pathol, 94pp. J Pediatr Orthop, 19pp. Pathophysiology of osteonecrosis of the jaw: Aguirre Canyadell aI. Thromb Haemost, 71pp. J Clin Invest, 94pp. Clin Chem, 32pp. enferedad
Am J Hematol, 44pp. Are you a health professional able to prescribe or dispense drugs? Conclusions Epidemiological and laboratory data from this group ofpediatric patients and from the control group do not supportthe hypothesis that a thrombogenesis defect couldbe the underlying cause of avascular necrosis of the hipjoint.
Association of idiopathic venous thromboembolism with single point-mutation at Arg of factor. Lancet,pp. Activated protein C resistance as an additional risk factor for thrombosis in protein C deficient families.
Enfermedad de Legg-Calve-Perthes | HCA Healthcare
Alterations in hemostasis and thrombosis were detectedin one patient who had moderate-to-light protein Sdeficiency. Blood, 87pp. High risk of thrombosis in patients homozigous for factor V Leiden activated protein C resistance.
Protein C and S deficiency, thrombofilia and hypofibrinolysis: Blood collection in strongly acid perthea anticoagulant used in a study of dietary influence of basal Tpa activity.